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Thank you for visiting nature. To obtain the best experience, we recommend you use a more up to date browser or turn off compatibility mode in Internet Explorer. Here, we show a novel effect of LCKD associated with the conflicting effects on these mice. Consumption of low-carbohydrate ketogenic diets LCKDs has been shown to have diverse beneficial effects in promoting health and alleviating pathologic conditions. For example, LCKDs exhibit antiepileptic effects, 1 promote weight loss in obesity, 2 and enhance control of type 2 diabetes mellitus. As a clinical trial demonstrated that restricting carbohydrate intake can reduce intrahepatic triglyceride levels, 7 increased research attention has focused on the use of LCKDs in treating liver steatosis. A number of studies using rodent models have analyzed the role of LCKDs in the development of liver steatosis. In addition, serum activity of lipoprotein lipase LPL, which mediates triglyceride uptake from VLDL into extrahepatic tissues, 18 was also down-regulated. These alterations in both strains were correlated with the conflicting liver steatosis phenotypes. The results of our study suggest that the transport of triglycerides via VLDL from the liver to extrahepatic tissues is inhibited by LCKD-induced hepatic VLDLR up-regulation under low LPL activity, whereas this inhibition of triglyceride transport is rescued under conditions of leptin deficiency.